The treatment I am recieving from doc

[Janice]

Talks about hidden parrasites

YouTube - Robert Rowen, MD Parasites Hidden...

[Janice]

YouTube - Dr. Robert Rowen, MD Vitamic C Amazing...

[JimDoe]

Originally Posted by Janice

No, I do not have any kind of alcahol dependency. The LDN is used for crohns disease, MS, chronic fatigue , aids, ect, ect ect. Some people in the morgelons world are very interested in this, they are currently on the side lines. If you get a chance google Low Dose Naltrexone. Very high doses deal with what you are talking about. Thanks, Janice

Now this LDN you bring up is very instersting. I recently went to emergcy room for spasms. This was when I had gone 8 days without eatting nothing but a bag of chips.. They ran blood testing on me. I was also coming off a period of drinking when the spasms hit. Test came back that I was depleted of Electrolytes. THey gave me an Iv line of something that was not glucose. I,ve seem glucose from an IV and it looked clear. This was syurpy grey stuff. Before I left a nurse asked me if I was going to drink when I got home. I said no. She sugested she give me something for achloe withdrawl ( a pill) would help me and feel better. I ok,d it and took the pill. Man did I feel better for almost a week. Morgs itching and bitting was gone for that time. I wonder now if it was LDN.

[Janice]

It very well could have been , but I wouldn't concentrate on that. Eat better, take your supplements, cut down on your drinking. You hold your health in your hands. You should be empowered, don't wander off. You can beat morgellons, but you have to take responsibiliy. Ya gotta get jiggy with it. Janice

[sammy]

Originally Posted by Janice

It very well could have been , but I wouldn't concentrate on that. Eat better, take your supplements, cut down on your drinking. You hold your health in your hands. You should be empowered, don't wander off. You can beat morgellons, but you have to take responsibiliy. Ya gotta get jiggy with it. Janice

Janice Do you have anything going on any more or are pretty clear of this stuff. My lesions have clearned up almost tks too the dudermm bandages but still hve the black specks and the white ones. started pouring vinger over my head after shower. sammy

[Janice]

I only have my right wrist involved now, but I had horrible leasons on my arms and legs. I had them so bad people would move away from me... they looked so bad. I am using desitine for the wrist and after the zinc was injected in my vit C IV the black specs poured out of my wrist (with the use of desitine.) The very sharp painful black hairs are gone. I think sad sack has a great idea with the dead sea salts.
I have another prescription of Mebendazole that I have put off taking but I will start it on monday. This helped me big time. It is an anti parasitic even if this thing is not a parrasite it worked for me. I don't know if you caught the utube video of the Doc in Alaska it's worth a look. he discusses hidden parasites. He believes in using more herbs for parasites as apposed to drugs. I will let you know if my wrist clears up compleatly after the Mebendazole. Janice

[Janice]

I wanted to break down my stool test results, I have a bit of Candida Albicans but twice as much Candida glabrata (torulopsis glabrata). It is listed as a potential pathogen on my test. I was wondering if any body that was tested came back with this?
Read this when you have a few minuets. The last paragraph is what is so interesting. This can involve the skin and scalp.

Candida glabrata
From Wikipedia, the free encyclopedia
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Candida glabrata
Scientific classification
Kingdom: Fungi
Phylum: Ascomycota
Subphylum: Saccharomycotina

Torulopsis glabrata

Candida glabrata is a haploid yeast of the genus Candida, previously known as Torulopsis glabrata. This species of yeast is non-dimorphic and no mating activity has been observed. Until recently, C. glabrata was thought to be a primarily non-pathogenic organism. However, with the ever increasing population of immunocompromised individuals, trends have shown C. glabrata to be a highly opportunistic pathogen of the urogenital tract, and of the bloodstream (Candidemia). It is especially prevalent in HIV positive people, and the elderly.

There are two widely cited potential virulence factors that contribute to the pathogenicity of C. glabrata. The first is a series of adhesins coded by the EPA (epithelial adhesin) genes. These genes, located in the subtelomeric region, can respond to environmental cues that allow them to be expressed en masse so the organism can adhere to biotic and abiotic surfaces in microbial mats. This is also the suspected mechanism by which C. glabrata forms microbial "biofilms" on urinary catheters, and less commonly in-dwelling IV catheters. It also causes problems with dental devices, such as dentures.
Contents
[hide]

* 1 Diagnosis
* 2 Treatment
* 3 References
* 4 External links

[edit] Diagnosis

Cultures are an effective method for identifying non-albicans vaginal infections. Urinalyses are less accurate in this process. The culture may take several days to grow, but the identification of the yeast species is quick once the yeast is isolated. Skin disease diagnosis is difficult, as cultures collected from swabs and biopsies will test negative for fungus and a special assessment is required. Listed under the 'Rare Diseases' database on the NIH web site, Tortulosis, or candida glabrata can also be found on the CDC's web site. Although listed as the second most virulent yeast after Candida Albicans, almost no information is available regarding treatment and identification. Although high mortality rates are listed, assessment of the critical nature of a glabrata infection is a gray area.
[edit] Treatment

A major phenotype and potential virulence factor that C. glabrata possesses is low-level intrinsic resistance to the azole drugs, which are the most commonly prescribed antifungal (antimycotic) drugs. These drugs, including fluconazole and ketoconazole, are "not effective in 15-20% of cases"* against C. glabrata. While some have said that the organism possesses an "innate" immunity to the drugs, it is more accurate to say that the organism possesses an evolved resistance to the drugs. It is still highly vulnerable to polyene drugs such as amphotericin B and nystatin, along with variable vulnerability to flucytosine and caspofungin. However doctors asked about amphotericin B indicate that this is a drug of last resort, in that this drug can often kill the patient while "curing" the fungal infection.

A first-line treatment for vaginal infections may be the use of Terconazole 7-day cream. Several courses may be needed. The cure-rate for this treatment is approximately 40%. Recurrences are common, causing chronic infections and spread to other areas such as skin and scalp. Blood infections might well be best assessed per symptoms if other areas are involved.

An experimental, but effective second-line treatment for chronic infections, is the use of boric acid. Compounding pharmacies can create boric acid vaginal suppositories. Use of Vitamin E oil may be used in conjunction to combat irritation. Amphotericin B vaginal suppositories have also been used in case studies to treat chronic infections, both symptomatic and asymptomatic. Borax and boric acid may be used for persistent scalp and skin infections. Very little information is available regarding treatment for Tortulosis glabrata.





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[Janice]

I have this as a potential pathogen on my test.
Please read when you have some time.

Proteus mirabilis is a Gram-negative, facultatively anaerobic bacterium. It shows swarming motility, and urease activity. P. mirabilis causes 90% of all 'Proteus' infections in humans.
Contents
[hide]

* 1 Diagnosis
* 2 Disease
* 3 Treatment
* 4 Characteristics
* 5 External links
* 6 Further reading

[edit] Diagnosis

An alkaline urine sample is a possible sign of P. mirabilis.

P. mirabilis can be diagnosed in the lab due to characteristic swarming motility, and inability to metabolize lactose (on a MacConkey agar plate, for example). Also P. mirabilis produces a very distinct odour.
[edit] Disease

This rod shaped bacterium has the ability to produce high levels of urease. Urease hydrolyzes urea to ammonia (NH3) and thus makes the urine more alkaline. If left untreated, the increased alkalinity can lead to the formation of crystals of struvite, calcium carbonate, and/or apatite. The bacteria can be found throughout the stones, and these bacteria lurking in the stones can reinitiate infection after antibiotic treatment. Once the stones develop, over time they may grow large enough to cause obstruction and renal failure. Proteus can also cause wound infections, septicemia and pneumonias, mostly in hospitalized patients.
[edit] Treatment

P. mirabilis is generally susceptible to most antibiotics apart from tetracycline, however 10%–20% of P. mirabilis strains are also resistant to first generation cephalosporins and ampicillins.
[edit] Characteristics

P. mirabilis can utilize urea and citrate. It can produce hydrogen sulfide gas, and forms clear films on growth media. It is motile, possessing peritrichous flagella, and is known for its swarming ability. It is commonly found in the intestinal tract of humans. P. mirabilis is not pathogenic in guinea pigs or chickens. Noteworthy is the ability of this species to inhibit growth of unrelated strains resulting in a macroscopically visible line of reduced bacterial growth where two swarming strains intersect. This line is named Dienes line after its discoverer Louis Dienes.

The micro-organism tests:

* Indole negative and Nitrate reductase positive (no gas bubbles produced)
* Methyl Red positive and Voges-Proskauer negative
* Catalase positive and Cytochrome Oxidase negative
* Phenylalanine Deaminase positive
* Tryptophan test- negative (-)
* Urea test- positive
* Casein test-negative
* Starch test- negative
* Hydrogen sulfide test- positive
* Citrate agar test- positive

[edit] External links

* "Proteus mirabilis and Urinary Tract Infection" [1].
* Proteus Genome Projects from Genomes OnLine Database
* Bacteria of the species Proteus mirabilis are widely distributed in soil and water in the natural environment. In humans, Proteus is found as part of the normal flora of the gut....from BioMed HTC - Proteus mirabilis BioMedHTC]

[adaption]

I was diagnosed with

-ehrlichiosis (attacks white blood cells)
-babesiosis (attacks red blood cells)
- 2 parasites in the gut (METAMATRIX DNA LAB....)
-1 nanomax
-1 unknown parasite

I also have a lot of skin weirdness, crawling in orifices, no lesions but
had some fibers, bugs, loads of brown specs. Sudden allergies to
all kinds of things. Sugar is totally off the menu. Diarrea/gas... all
part of the picture I guess.

I am afraid to take something that increases the oxygen, because the
babesiosis likes oxygen.... I wish there was a doctor who understood
the links between these things, who could say, yes, you can take this, but
also take this other thing at the same time, etc.

I would go to a sauna with FIR, but there isn't one nearby ... and I'm too
wiped out to travel right now.... I was doing pretty well, after 3 weeks
with a healer, but then stress at work got me whacked again...

[Janice]

I would bet... we all have these, or some of these bacteria and yeast infections in common. Is this combination of things... "morgellons" ?







itrobacter freundii are commonly found in the environment, mainly in soil, water, and sewages. They are an indicator of potential contamination of water. They are also found on different organs of diseased animals, including mammals, birds, reptiles, and amphibians [1]. They are not known to interact with other organims.

In the environment, C. freundii can convert nitrate or the ammonium ion (which is a nitrogen atom combined with four hydrogen atoms) to nitrite; this reaction occurs in the environment as well as within the digestive tract of humans and other animals [3]. After it converts nitrate to nitrite in the environment, the nitrite is converted to nitrogen, and this final step completes the nitrogen cycle in the earth's atmosphere, which is made up of 85% nitrogen [3]. This organism's ecological role not only includes its important role in the nitrogen cycle, because it can also accumulate uranium (which is the basic material for nuclear technology) by building phosphate complexes [3].

Citrobacter freundii has also been investigated for biodegradation of tannic acid used in tannerys [3].
Pathology

As an opportunistic pathogen, Citrobacter freundii is often the cause of significant opportunistic infections, meaning that it does not generally cause disease in healthy human hosts. They only affect patients with a weak immune system, signifying that they need an "opportunity" to infect the person [2]. Therefore, in patients with a suppressed immune system, Citrobacter species are known to cause a wide variety of nosocomial infections of the respiratory tract, urinary tract, and the blood [2]. Hepatic, biliary and pancreatic disease are also common diseases that are caused by C. freundii. The biliary tract is the most common site of infection by the C. freundii bacilli [9].

One fatal disease that C. freundii has been associated with is neonatal meningitis. Neonatal meningitis is the inflammation of the meninges (the system of membranes which surround the CNS) due to bacterial invasion [10]. The mortality rate of Citrobacter meningitis is unacceptably high, with death rates of patients ranging from 25 to 50 %. Moreover, serious neurological problems still persist in 75% of survivors. In this disease, Citrobacter freundii is able to penetrate the blood-brain barrier that consists of the choroid plexus epithelium and the brain capillary endothelium [10].

Tests performed by Badger et. al in the article “Citrobacter freundii Invades and Replicates in Human Brain Microvascular Endothelial Cells”suggest that bacterial proliferation of C. freundii takes place at the intracellular level, which had been contrary to the general scientific thought. The findings indicate that C. freundii traverses vacuoles, replicates and is released into the basolateral side of the human brain microvascular endothelial cells (HBMEC) in order to cross the blood-brain barrier. Further analysis may potentially allow for therapeutic strategies to treat infections. There is still no therapeutic treatment available [22].

Certain diseases studied in trout and cyprinids are also caused by C. freundii. C. freundii causes abnormal inflammatory changes in the intestine of trout and inflammatory and necrotic changes in the internal organs of cyprinids. The illness was discovered by means of artificial infection with a pure culture of C. freundii. This discovery established C. freundii as a cause of fish disease [11].

In a case study by the Journal of Medical Microbiology, a patient developed peritonitis and tunnel infection due to Citrobacter freundii which is uncommon. The patient was on continuous ambulatory peritoneal dialysis. Usually the causing agents are gram-positive micro-organisms, particularly Staphylococcus aureus and Staphyloccus edpidermis. Also there are no known reports of tunnel infection due to C. freundii. Initial antibiotic therapy did not work and the infection continued to persist until the catheter was removed. This is clinically significant because Citrobacter Freundii show different antibiotic susceptibility which is why initial therapy was not successful. The patient did not respond to treatment until the catheter was removed showing Citrobacter freundii are opportunistic pathogens that affect hospitalized and immunocompromised patients [18].
Application to Biotechnology