Evidence pointing towards immune system hyper-activity

[tcmgpt13]

Kritts, your link does not seem to be the answer to dissolving chitin in the body, but it may show the required chemical steps to achieve that in a drug. Here are some problems scientists are running into in treating the larvae which carries one form of malaria parasites. Apparently the drug used does not effect chitin at all stages. Correct timing of wormers seems to be crucial for effective control of parasites, so it is easy to see that if a chitin type parasite is involved in some who have morgellons then it would also be necessary to figure out exactly in what stage the wormer or drug would work to control them. The drug spoken of in this link is diflubenzuron which I assume must be sprayed on waters where the mosquito larva are before hatching into the adult mosquito which carries the malaria parasite to man:

Comparative studies on effects of three chitin synthesis inhibitors on common malaria mosquito (Diptera: Culicidae)

Titre du document / Document title
Comparative studies on effects of three chitin synthesis inhibitors on common malaria mosquito (Diptera: Culicidae)
Auteur(s) / Author(s)
KUN YAN ZHU (1) ; HEISE Stephanie (1) ; JIANZHEN ZHANG (1) ; ANDERSON Troy D. (1) ; STARKEY Sharon R. (1) ;
Affiliation(s) du ou des auteurs / Author(s) Affiliation(s)
(1) Department of Entomology, 123 Waters Hall, Kansas State University, Manhattan, KS 66506, ETATS-UNIS
Résumé / Abstract
Toxicities of three chitin synthesis inhibitors (diflubenzuron, nikkomycin Z and polyoxin D) were evaluated using second instars of the common malaria mosquito, Anopheles quadrimaculatus Say (Diptera: Culicidae). Neither nikkomycin Z nor polyoxin D at 50 μg/liter caused significant larval mortality, although they reduced the body weight of the survivors by 20.5 and 33.8%, respectively, in 48 h. In contrast, exposures of the larvae to diflubenzuron at 12.5 μg/liter for 48 h resulted in 86.7% larval mortality and reduced the body weight of the survivors by 29.1%. Exposure of the pupae (<12 h old) to diflubenzuron at 100 μg/liter for 48 h caused 18.9% pupal mortality and consequently reduced the adult emergence by 24.7% from the surviving pupae. Furthermore, exposure of third instars to diflubenzuron at 4, 20, 100, and 500 μg/liter for 24 h resulted in the reduction of larval chitin contents by 4.25, 33.2, 35.2, and 57.7%, respectively. Such an effect seemed to be associated with only cuticular chitin synthesis because the same exposures did not significantly affect chitin contents in the guts. Our results indicated that diflubenzuron was highly toxic to second instars by not only causing high larval mortality but also by affecting their growth. Diflubenzuron was also fairly toxic to pupae by not only causing pupal mortality but also affecting the adult emergence. Our results suggest that diflubenzuron might affect only chitin synthesis in the cuticle but not in the peritrophic matrix, which is probably due to diflubenzuron's direct contact to mosquito larvae in water, slow distribution in insect body, rapid degradation in the insect gut, or a combination.

[Kritters]

I love it TC....

You are amazingly detailed. I jump onto something and then jump quickly to the next. That's why I love you. Of course, not only why...you are a really incredible person.

I will throw it out and because I have limited attention span, you can check it out!

xoxo
Kritts