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| Morgellons Theories & Speculations Discussion on Theories and Speculations on Morgellons |
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| Duplicate post from 'Paper Thread': Howie - we are 'on' this P.a., already. We have some very, very interesting news about this particular bacteria that I will be publishing soon. Kat and I now have the capability to look at the microscopic slides and data BEFORE we publish, to more closely scrutinize what is said. I have a new, YouTube out that we believe shows this bacteria reproducing and it in several stages. At the beginning, it shows it as like a deflated balloon and behind it - it has left a trail of biofilm and 'babies'. I believe this is just one of many ways this bacteria reproduces, I have many photos of it using various methods of reproduction, these methods have not been verified yet. At the end of the video - it shows an immature coccus seed that is 'sitting' on a more mature 'fuzzier' one that I believe is in a more advanced stage. I did something unusual to my ear to finally get this pathogen to reveal itself in this manner, cultured June 5th, after 6 weeks of diflucan (no fungus present), and one week of Levaquin - only 2 or so 'fibers' in the dish, this video represents 3 weeks growth, photographed at 300x: Howie - How did YOU come to your conclusion that this bacteria might be involved?... Last edited by -----------; June 27th, 2009 at 10:41 AM. |
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| Pseudomonas aeruginosa OK, what's going on? Are we introducing a new pathogen... no, this guy has been here all along. Due to the nature of what we're dealing with - I'm going to make some mistakes. I'm now thinking that this is what was earlier identified as 'Crypto' and then changed to 'Peridiscaceae'. That we've been looking at a bacteria all this time. Input please? I added an experimental solution to my ear right before I cultured it to see what effect this solution would have on the growth. This caused all the 'others' to be missing, or the medicines I had just taken caused this? and allowed this one main, what I have been calling a 'seed' pathogen to be primary in the dish. This pathogen starts out as a salt crystal. It is an exact match to the wikipedia dish growth photo. That by the other factors being in the dish previously, what it appeared to be visually, (the white circular connected growths), was masked by the other pathogen's influences? Wikipedia photo: P. aeruginosa on an XLD agar plate. I took this dish to an ENT yesterday, already suspecting P.a. from a visual match, which he verified that it was this pathogen by visual inspection, it has a distinct odor, he said, he looked and seemed very familiar with this pathogen, especially, recognized the odor (smells like a musty basement in the dish) and is treating me with internal and topical ciproflaxin, diflucan and an over-the-counter topical, anti-fungal for athlete's feet. The solution also allowed its distinct odor to be noticeable, which in the past - was not. I have just finished 16 days of Biaxin and Quensyl, in which I cultured once again, prior to the Ciproflaxin, which I just started. The results of the Biaxin/Quensyl culture will be noted, also. To be specific, the ENT did not send my dish to a lab, nor did he culture my ear. I have no lab documentation to verify these findings, he requested a follow-up visit in 3 weeks, I am being treated for a Pseudomonas aeruginosa infection of my ear. Last edited by -----------; June 27th, 2009 at 04:36 PM. |
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| What this solution that I put on my ear did (and/or medicines taken) was cause this particular pathogen to become isolated and to speed up its 'growth'? processes - it shows it going through its various stages much quicker than in previous videos. Last edited by -----------; June 27th, 2009 at 11:23 AM. |
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| Like all of you, I've struggled for so long to make sense of this illness and have spent lots of time researching possibilities. None of the ideas I came across really answered the questions and, as time went on, I realized that what I thought were worms under my skin might actually be just sensations and not real worms. To know now that it is quite probably just a bacteria that's "playing" in my cilia (nerve endings) is a little more comforting than thinking I have worms in my skin! It was just a fluke that I came across this bacteria - i didn't discover it on the internet but in a small book from the 1960's that explored different species of bacteria. The problem with the internet is that sometimes there's just too much info to sift through and I've always found the best way to get to the answer is to simplify the approach. (I love libraries.) The symptoms ascribed in the book seemed to fit to a tee. As I explored it on the internet and found all the ways various strains are used, the symptoms fit, the nursing connection fit, the cluster outbreaks fit and even creepy things coming out of me fit. This bacteria makes tons of protein, it makes slime, it can even make algae, it protects itself with a biofilm and would attract insects that eat bacteria. I concluded that perhaps we're all not necessarily "infected" as in high fever, we're all going to die from this, just perhaps that we're not feeding it what it wants and it is reacting. I think there may be more danger from the parasitic organisms that are attracted to us because of it and the threat they pose to our long term health. The responsible thing for all of us to do is to assist in finding answers before this illness escalates out of control. I think that bioremediation is a necessary evil because we've been so careless about caring for our planet. And I don't see the problem with reintroducing normal fungi and bacteria into soil and waters to make them healthy. My concern is who's monitoring all this and who's monitoring all the pesticides and fertilizers to make sure there isn't some new contamination occurring at bioremediation sites. There are idiots in every industry and all it would take to have a huge health disaster would be allowing an unqualified company to perform bioremediation work using these bacterial strains or having genetically modified organisms introduced and causing worse contamination. Bacteria are known to join forces with other organisms in order to preserve their own survival. Who knows if the EPA even keeps track of any of this... Meanwhile, I'll stick to filtered water as much as I can. I'm so glad you found my ideas helpful. |
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| Yes, Howie, very helpful! Sometimes, things come together like this - for a reason... If what I'm showing isn't P.a.... I have something very darn similiar, we need to look at this and see what all it's capable of because this is how some of us need to be treating ourselves in order to get better. Last edited by -----------; June 27th, 2009 at 03:03 PM. |
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| Hey Howie! Thanks! I wanted to add a little more information here about PA..could be that this has been already mentioned before... P. aeruginosa secretes a variety of pigments, including pyocyanin (blue-green), fluorescein (yellow-green and fluorescent, now also known as pyoverdin), and pyorubin (red-brown). P. aeruginosa is often preliminarily identified by its pearlescent appearance and grape-like or tortilla-like odour in vitro. Definitive clinical identification of P. aeruginosa often includes identifying the production of both pyocyanin and fluorescein, as well as its ability to grow at 42°C. P. aeruginosa is capable of growth in diesel and jet fuel, where it is known as a hydrocarbon-utilizing microorganism (or "HUM bug"), causing microbial corrosion** It creates dark gellish mats sometimes improperly called "algae" because of their appearance. **does this mean PA can survive even in fuel/air mix? Fuel residues in our water systems...?? An opportunistic pathogen of immunocompromised individuals, P. aeruginosa typically infects the pulmonary tract, urinary tract, burns, wounds, and also causes other blood infections. It is the most common cause of infections of burn injuries and of the external ear (otitis externa), and is the most frequent colonizer of medical devices (e.g., catheters). Pseudomonas can, in rare circumstances, cause community-acquired pneumonias,as well as ventilator-associated pneumonias, being one of the most common agents isolated in several studies. Pyocyanin is a virulence factor of the bacteria and has been known to cause death in C. elegans by oxidative stress. However, research indicates that salicylic acid can inhibit pyocyanin production.One in ten hospital-acquired infections are from Pseudomonas. Cystic fibrosis patients are also predisposed to P. aeruginosa infection of the lungs. P. aeruginosa may also be a common cause of "hot-tub rash" (dermatitis), caused by lack of proper, periodic attention to water quality. The most common cause of burn infections is P. aeruginosa. Pseudomonas is also a common cause of post-operative infection in radial keratotomy surgery patients. The organism is also associated with the skin lesion ecthyma gangrenosum. It has been found that with low phosphate levels, P. aeruginosa is activated from benign symbiont to express lethal toxins inside the intestinal tract and severely damage or kill the host, which can be mitigated by providing excess phosphate instead of antibiotics. Kam%**, didn't you tell me that the 'sollution' you used on your ear contained phosphates? Maybe this is/was the reason that PA reacted so quickly? like you already said? YES!! With plants, P. aeruginosa induces symptoms of soft rot with Arabidopsis thaliana (Thale cress) and Lactuca sativa (Lettuce). It is a powerful pathogen with Arabidopsis and with some animals: Caenorhabditis elegans,Drosophila and Galleria mellonella. The associations of virulence factors are the same for vegetal and animal infections. And..did you know that experiments were/are done with PA and nematodes classified as Caenorhabditis elegans?? Flies ? and moths? And that this type of Nematode mixed with PA was brought along for experiments on the Space Shuttle Columbia which 'crashed' 2003? A group of small (1 mm adult) Caenorhabditis elegans worms, living in petri dishes enclosed in aluminium canisters, survived re-entry and impact with the ground and were recovered weeks after the disaster. The culture was verified as still alive on April 28, 2003. They were part of a Biological Research in canisters experiment designed to study the effect of weightlessness on physiology. It might mean nothing...but keeps me sort of thinking...Nano gel..remember? Kat Last edited by Katinka; June 27th, 2009 at 03:12 PM. |
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| ASPIRIN EXPERIMENT "However, research indicates that salicylic acid can inhibit pyocyanin production.One in ten hospital-acquired infections are from Pseudomonas." When I was reading up on PA, I read this so, I took a small glass of water that my ear would fit into exactly and put it in the microwave for 1 minute and made the water hot, put 4 aspirins in the water, dissolved it and took 2 aspirins at the same time - let it cool down and then soaked my ear in this glass of water for as long as I could hold this position, approx. 10 minutes. My ear showed improvement in this short amount of time. I haven't repeated this experiment, I only did it once - I am now putting these other medicines on it. We might want to try some aspirin/water solution testing to see if this helps with lesions? The Andromeda Strain? |
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| These pictures look quite similar to what shows in Kam's dish: ![]() ![]() The Same but Different: 30 times magnified image of colonies from four different strains of the bacterium Pseudomonas aeruginosa (a human pathogen under some conditions) as it is grown under routine laboratory conditions. Even though they are all the same bacteria it is clearly visible how the bacteria have adapted their morphology to survive in their respective environments. Kat |
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| Of course, this one pathogen isn't all there is... from what we've seen - everything/anything can get 'caught up in' this biofilm PA emits. We know that the key to getting well is to eliminate the biofilm, it's the most difficult part of this disease to rid. It's the womb for everything else! |
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| agrobacterium, antibiotic resistance, biofilm, manuka honey, medical grade honey, morgellons, pseudomonas aeruginosa, silver bandaids |
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